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We concur with Bornstein and Matarazzo s (1984) plea for a comprehensive prospective study that, at the very least, controls for a number of potentially important lesion-related variables as a means of delineating the role of gender in V P discrepancies in patients with unilateral brain damage. However, in the absence of such a landmark study and of a large-scale retrospective study with enough patients in the sample to permit statistical control of such potentially relevant variables...by involving a number of large medical centers in a single, coordinated effort (p. 709), we must try to understand the variable of gender as best we can based on the considerable database available. We disagree emphatically with Jacklin s (1989) strong suggestion to stop conducting research on gender-related cognitive differences. Meta-analysis (Bornstein & Matarazzo, 1984; Inglis & Lawson, 1982; Snow et al., 1986; Turkheimer & Farace, 1992) provides one useful research technique. Another is to compare the V P differences of males and females only from those reasonably well-controlled investigations that included both sexes in approximately equal numbers. Inglis and Lawson (1982) did compare V P discrepancies for separate groups of male and female patients, combining data from several studies. However, they failed to match the male and female samples on any pertinent variables. For example, their aggregated sample of male patients included data from studies comprising only males. Consequently, the sample of 170

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Table 8.14 presents data from nine studies that included males and females in approximately equal proportions. Most of these studies were extremely well designed, controlling for differences between the genders on variables like age, education, nature of lesion, and recency of lesion. Despite Snow et al. s (1986) cautions to the contrary, data were combined across instruments, as the Wechsler-Bellevue, WAIS, and WAIS-R were all used for one or more studies. The instrument given has, nonetheless, been held constant because the same Wechsler test was administered to men and women in each separate study. To a reasonable degree, the variables of concern to Bornstein and Matarazzo (1984) and Snow et al. (1986) were controlled in the male female comparisons presented in Table 8.14. The neurodiagnostic criteria that were used in each study, whether primitive or high-tech, were applied equally to males and females. Similarly, variables such as age, education, recency of lesion, and etiology were not appreciably different for the men and women within each separate investigation. Therefore, the data shown in Table 8.14 come as close as is presently feasible to the type of coordinated effort that Bornstein and Matarazzo have urged. As shown in Table 8.14, each gender is represented by more than 125 patients with lefthemisphere lesions and more than 145 patients with right-hemisphere lesions; data are based on

276 women and 296 men with unilateral brain damage. Taken separately, six of the nine studies showed apparent gender differences in the V P discrepancies for patients with lesions in each hemisphere; only the investigations by Snow and Sheese (1985), Bornstein (1984), and Whelan and Walker (1988) failed to discern meaningful gender-related findings. Taken together, P > V was 5.6 points for males with left lesions but only 0.9 points for females with left lesions. Similarly, patients with lesions in the right hemisphere evidenced larger V > P differences if they were male (13.2 points) than if they were female (7.2 points). We believe that the accumulated data in Table 8.14 show strong support for the alleged gender-related interaction between side of lesion and direction of V P difference. For both genders, damage to the right hemisphere produced more striking V P discrepancies than damage to the left hemisphere. But for males, the V > P of about 13 points for right-lesioned patients is nearly twice the value of 7 points for females. The 51 2-point P > V difference for left-damaged adult males does not measure up to the opposite discrepancy for right-damaged adult males, but the observed difference still gives good support to the predicted hypothesis of depressed Verbal IQs for patients with lesions in their left hemispheres. For females, however, the P > V discrepancy of less than 1 point for those with lefthemisphere lesions does not support the old hypothesis. In sum, women apparently have a different cerebral organization than men, and may differ in the asymmetry of their brains. However, the reason for the interaction with gender is far less obvious than the fact of it.

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